8.2.0.0 Neona­tal Di­a­betes

Di­a­betes oc­cur­ring under 6 months of age is termed “neona­tal” or “con­gen­i­tal” di­a­betes, and about 80–85% of cases can be found to have an un­der­ly­ing mono­genic cause (120). Neona­tal di­a­betes oc­curs much less often after 6 months of age, where­as au­toim­mune type 1 di­a­betes rarely oc­curs be­fore 6 months of age. Neona­tal di­a­betes can ei­ther be tran­sient or per­ma­nent. Tran­sient di­a­betes is most often due to over­ex­pres­sion of genes on chro­mo­some 6q24, is recur­rent in about half of cases, and may be treat­able with med­i­ca­tions other than in­sulin. Per­ma­nent neona­tal di­a­betes is most com­monly due to au­to­so­mal dom­i­nant mu­ta­tions in the genes en­cod­ing the Kir6.2 sub­unit (KCNJ11) and SUR1 sub­unit (ABCC8) of the β-cell KATP chan­nel. Cor­rect di­ag­no­sis has crit­i­cal im­pli­ca­tions be­cause most pa­tients with KATP-‍re­lat­ed neona­tal di­a­betes will ex­hib­it im­proved glycemic con­trol when treat­ed with high-‍dose oral sul­fony­lureas in­stead of in­sulin. In­sulin gene (INS) mu­ta­tions are the sec­ond most com­mon cause of per­ma­nent neona­tal di­a­betes, and, while in­ten­sive in­sulin man­age­ment is cur­rently the pre­ferred treat­ment strat­e­gy, there are im­por­tant ge­net­ic con­sid­er­a­tions, as most of the mu­ta­tions that cause di­a­betes are dom­i­nantly in­her­it­ed.